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Comparison of kidney transplant survival between patients treated with cyclosporine and those treated with azathioprine and antithymocyte globulin.
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It is also used to prevent rejection of transplanted organs, such as the kidney, heart, and liver.
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Although concomitant administration of diclofenac does not affect blood concentrations of Cyclosporine, it has been associated with approximate doubling of diclofenac blood concentrations and occasional reports of reversible decreases in renal function.
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PREGNANCY and BREAST-FEEDING: Cyclosporine may cause harm to the fetus.
However HDL, LDL and cyclosporine serum levels had no significant differences by both of swallowing and chewing routes.
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Therefore, not only is the regimen effective, but there are some patients who continue to tolerate it 5 years out.
Cyclosporine, the active ingredient in Cyclosporine Oral Solution USP MODIFIED, can cause nephrotoxicity and hypertension (see ) and the risk increases with increasing dose and duration of therapy.
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Activation of T-lymphocytes results in activated calcineurin, which dephosphorylates inactive nuclear factor (NFAT).
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The above data demonstrate that the types of adverse events observed in multicenter controlled trials in renal, cardiac, and hepatic transplant patients are qualitatively similar except for those that are unique to the specific organ involved.
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The finding of acute reversible nephrotoxicity and nephrotoxicity in nonrenal transplants has since led to the widely accepted view that there is a chronic more irreversible component to this agent as well.
The treatment reversed agerelated loss of fur, boosted the rodents vitality and improved their kidney function, according to a new report.
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Antibiotic therapy was not initiated during the study unless indicated -- for example, for pulmonary or urinary tract infection.
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During the eight weeks of cyclosporine treatment, 7 of the 16 patients had undetectable levels of the drug in their blood (lower limit of detection, 10 ng per milliliter).
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The changes in erosion, erythema, reticulation, and symptoms for each patient were examined by comparing the scores before therapy and at the final (eight-week) evaluation in the patients receiving either cyclosporine or vehicle, with a two-tailed Mann-Whitney test.
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Although magnesium-depletion studies in normal subjects suggest that hypomagnesemia is associated with neurologic disorders, multiple factors, including hypertension, high dose methylprednisolone, hypocholesterolemia, and nephrotoxicity associated with high plasma concentrations of Cyclosporine appear to be related to the neurological manifestations of Cyclosporine toxicity.
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El informe aparece en la edicin en lnea del 27 de marzo de la revista JAMA Internal Medicine.
Cyclosporine has been shown to be embryo- and fetotoxic in rats and rabbits following oral administration at maternally toxic doses.
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